Papillary sclerosing lesion breast, V-ar putea interesa


Congenital anomalies of the heart and vessels 10 Heart malformations are determined by various factors, some with severe movement disorders and oxygen that are incompatible with life, other compatible although initially not generally allow a long-term survival.

They occur in animals as lack or excess malformations by malformations of position, or structural alterations septs or the heart valves. Shows theoretical and practical importance: Acardia total lack of heartlack of closing the pericardial sac, diplocardia double heart multiplicitas cordis multiple cordsdextrocardia heart on the right side of the mediastinumcardiac ectopia presence of heart in the cervical region, pectoral or abdominalpapillary sclerosing lesion breast.

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Septs structural defects are common to all species. The total or partial lack thereof, determines the appearance of cords bilocular right or left respectively upper and lower or bilocular cords biventricular, trilocular, biatriale or malformations incompatible with life.

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Persistence of the oval hole foramen ovale persistence after birth is located in interarterial septum and favors the mixing of the arterial with venous blood eventually causing death by hypoxia. In the papillary sclerosing lesion breast of all valves, especially in the atrioventricular ones, can be found congenital hematic cyst with the diameter to an inch.

Pericardium injuries Dystrophic lesions of the pericardium is of less importance in mammals, while in birds they produce irreversible changes sometimes incompatible with life. Serous atrophy of the pericardium occurs in older animals, in the chronic wasting disease. Adipose tissue in the heart is replaced with a substantial amount of transudate. The lesion is also known as gelatinous edema papillary sclerosing lesion breast "ex edema vacuo".

Macroscopic instead of the fat tissue there is a yellow gelatinous mass-glassy, wet on section. Serous atrophy has practical importance in veterinary checks for meat, it indicates a state of cachexia of the slaughtered animal.

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Fat infiltration of epicardium is the opposite of serous atrophy. It is characterized by excessive storage of fat in this stage, and sometimes fat necrosis.

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It is found mainly in animals subjected to the process of fattening cattle, pigs, poultry. Melanosis pericardial is a congenital process which occurs in calves, lambs, etc.

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Pericardial Gout is found in birds, papillary sclerosing lesion breast the expression status of general uric diathesis. Macroscopic, first,by the transparency of the pericardium is noticed in the pericardial cavity the presence of a white deposit like chalk more or less adherent to pericardium. Epicardium appears sprinkled with white paint.

Microscopic reveals acicular crystals arranged in rosettes or amorphous masses uropurinice. In longest developments on the outskirts chisturi giardia lamblia adulti uric deposits a lymphohystocytar mesenchymal reaction is observed with the appearance of giant cells, ultimately resulting in foreign body granulomas granulomatous pericarditis.

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Installing the inflammatory process is possible only in cases where the bird holding a long "uric poisoning", facilitating the emergence and development of granulomatous inflammation. Changes in blood and lymphatic circulation Pericardial bleeding, manifested by bruising papillary sclerosing lesion breast suffusions, characterize the poisoning, being the consequence of increased fragility of the capillary basement membrane. Macroscopically, on the surface of the pericardium and especially to the heart epicardium indicate the presence of small reddish points distributed over the deposit of fat and coronary sulcus.

Such lesions are found in diseases like: - bacterial septic emice: anthrax, pasteurellosis, streptococci; - virotic: classical and African swine fever, Newcastle disease; - toxic: warfarin poisoning or with derivatives.

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It is found in all papillary sclerosing lesion breast, but especially horses, pigs and dogs. Effusion is the accumulation of the transudation into the pericardial cavity. Pericardium and epicardium is shiny and smooth. It occurs in all species, with greater practical importance in pigs and birds. Chylopericardium is the accumulation in the pericardial cavity of lymph as a result of breakagethe thoracic duct Macroscopic, the presence of a lipid-rich milky white liquid.

All "pericardial collections" described above, have a bad prognosis as may be the cause of sudden death in the so-called "cardiac tamponade". Inflammation of the pericardium Inflammation of the pericardium are known under the generic name of pericarditis, it affects both blades of the pericardium and epicardium, the inflammatory process affects only the visceral foil. Are commonly found in cattle, poultry, swine, horses and carnivores are produced by toxic agents, infections and trauma.

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From an evolutionary standpoint pericarditis may be acute, subacute and chronic. Pathologically are classified pericarditis: necrotic, serous, fibrinous, purulent, traumatic, Ihor, fibrotic and granulomatous. Necrotic pericarditis is common in lambs visceral necrobacillosis s being linked with lung necrotic foci.

Macroscopic in the parietal serous pericardium are present outbreaks of gray-yellow, isolated or confluent, looking brittle, dry, of various sizes, surrounded by line bleeding in acute developments, less white in chronic developments. Serous Pericarditis is an inflammation observed in the evolution of acute septicaemic disease avian cholera, hydropericardium ruminants, etc. Parietal and visceral pericardial blades are congested, shiny and wet. Microscopic is observed vacuolation and desquamation of the mesothelial cells, congestion and edema subepicardic, leucodiapedesis.

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Serous pericarditis, stopped at this stage, has a benign evolution, being among the few sores that heal by "restitutio ad integrum". In general, it evolves into a serofibrinous inflammation and fibrinous eg papillary sclerosing lesion breast choleraor the serohemorhagic inflammation eg coal emphysematous.

Fibrinous pericarditis occurs in a number of infectious diseases and in the first phase of traumatic pericarditis in cattle. It is papillary sclerosing lesion breast by the appearance and accumulation of fibrinous exudate in the pericardial cavity. Macroscopic, serous pericardium strips are opaque, thickened, rough, due to the deposition of papillary sclerosing lesion breast on their surface.

Pericardial cavity is enlarged and contains appreciable quantities parazitii romania trezeste-te yellowish fibrin. Fibrinous exudate can unite the two skins of the serous pericardium, resulting in adhesions that can be easily removed. In the fibrin mass can accumulate fibrin exudate serous or purulent or hemorrhagic. In relation to the predominance of one of these exudates, mixed pericarditis occurs serofibrinous, fibrinohaemorrhagic or fibrinopurulent, which are forms of transition from one lesion to another.

These specific pathological aspects are specific of acute days pericarditis.

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In developments longer resorption occurs partially liquid, organizing fibrinous exudate on account of conjunctive tissue subepicardic, ending with the appearance on the epicardial surface of some gray-white filaments, or villi-like hairs, printing the heart a matter of "villous heart" or "hairy heart. In longer developments papillary sclerosing lesion breast there is organization, there is connective tissue proliferation and capillary forming the junction between the parietal and visceral tabs of the serous pericardium.

Appearing tissue is scar tissue that creates synechiae epicardo-pericardial ,pathologic characteristic aspect of the fibrous pericarditis.

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Purulent or suppurative papillary sclerosing lesion breast occurs most often as a further unfavorable fibrinous pericarditis, or occurs as a metastatic purulent pericarditis in Gurm and piosepticemia foals, piglets piobacillosis.

Macroscopic the pericardial sac is increased in volume to the palpation ,can be feel sometimes a liquid consistency, fluctuating, sometimes more viscous. On section, the pericardiumblades are much thickened and in the pericardial cavity is found varying amounts of purulent exudate.

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Microscopic pericardial blades are covered with purulent exudate, mesothelium is flaky or turned in piophage, subserous connective tissue is heavily infiltrated with granulocytes, capillaries are ectasies and with an intense leucodiapedesis process. Purulent pericarditis is a very serious, often being lethal. It is known as the piopericard or pericardial empyema.

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Traumatic pericarditis commonly found in cattle, is determined by the penetration of sharp metal body of the network through the diaphragm into the heart. Foreign bodies carry with them a wide range of germs including pyogenic and anaerobic. Following the initial pericardial traum first occure a serofibrinous inflammation then purulent, the last form being able to turn into gangrenous pericarditis.

These forms are dependent by the circulated microbial flora. Macroscopic appearance is dependent on the morphological form in which is surprised the evolution of the traumatic pericarditis.

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In general, evolution is chronic, proliferation processes dominated conjunctiva, papillary sclerosing lesion breast the advent of synechiae between the two pericardium skins fibroadhesive pericarditis.

Pericarditis gangrenous or Ihor is, in most cases, the consequence of traumatic pericarditis, or occurs as a complication of purulent pericarditis. Macroscopic volume of the pericardial sac is increased occurs by gas pneumopericard and by ihor exudate.

When the he pericardial cavity is opened a distinct odor of gangrene is smelled. Ihor effusion is greenishbrown fluid, gastric cancer her2 scoring, and sometimes being able to detect foreign bodies or fragments thereof.

Pericarditis is gangrenous and sapremia death by heart failure that it determines.

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Pericarditis fibrous hyperplastic or fibroblast is an evolving chronic productive inflammation, the result, in general, of the exudative pericarditis: serous, fibrinous or purulent. Fibrous pericarditis occurs in all species pasteurellosis, salmonellosis in pigs and poultry in the hemofilosis of swine mycoplasmosis in colibacillosis and avian mycoplasmosis etc. Macroscopic, it appears pericardial thickening due to a proliferative process with the starting base in the the subseries pericardial connective tissue fibrous pericarditis simple.

Thickened pericardium sometimes has some hairs on the surface pericarditis viloasa. The two blades of the pericardium will stick together by clamps and synechiae, taking the appearance of "fibro-adhesive pericarditis" common in pigs.